Ki sentòm yo ak kòz atak kè

Koulye a, yon jou atak kè se yon maladi trè komen nan sosyete a. Li komen nan laj fin vye granmoun ak jèn moun menm jan an. Se konsa, tout moun nan rada maladi kè menm ogmante li nan timoun tou. Se konsa, nou pral aprann sou ki jan atak kè se kòz ak ki sentòm yo nan atak kè oswa ensifizans kadyak. Se konsa, kite chèche konnen tout detay pwoblèm kè ki gen rapò.

Ki sa ki fonksyon yo ak ki jan li fonksyone?

Kè a ponpe san nan kò a pote oksijèn ak eleman nitritif nan tout selil nan kò a pou yo ka siviv epi fè travay yo.. San an tou pote pwodwi dechè soti nan selil yo nan ògàn yo ki debarase m de yon fatra tankou ou ren, kè ou se yon ponp doub.

• La bò dwat ponpe san nan poumon ou kote san an pran oksijèn epi li retounen sou bò gòch kè a..
• La vantrikul gòch Lè sa a, ponpe san nan kò ou atravè gwo atè ki rele aort la. Kòm san an sikile oksijèn deplase soti nan san ou nan selil yo nan tout kò ou.

San ki pa gen oksijèn retounen sou bò dwat kè a nan venn ou. Pwosesis sa a rive ak chak batman kè. Kè a li menm bezwen yon rezèv konstan nan oksijèn. Se oksijèn ki pi rich nan san ki pote nan atè yo. Atè sa yo jwenn sou sifas kè a.

Ki sa ki lakòz atak kè ak echèk?

Genyen de atè prensipal atè prensipal ke yo rekonèt kòm atè kowonè

1. Atè prensipal kowonè gòch la
2. Atè kowonè dwat la

Lè sa a, de atè sa yo divize an anpil branch ki pi piti ki antre nan misk kè a. Nenpòt anomali nan atè kowonè ka lakòz koule oksijèn ak eleman nitritif redwi nan kè a, ki ka mennen nan yon atak kè ak pètèt lanmò. Sa a ka lakòz atè a etwat oswa vin bloke yon fòm ateroskleroz. Sa a se kòz ki pi komen nan maladi kè. Maladi kowonè ke yo rele tou maladi atè kowonè oswa COPD se yon redi nan atè yo ki te koze pa akimilasyon nan depo gra., ki souvan rele Plak. Sa a akimilasyon plak rive anpil nan kouch ki pi anndan an nan atè yo kowonè.

Depo gra yo ka pwodui nan anfans epi kontinye epesè ak elaji pandan lavi. Kòm plak la bati moute atè a etwat ak sikilasyon san vin ba nan misk kè a. Lè sikilasyon san diminye, ou diminye koule nan oksijèn epi eleman nitritif nan kè a. Lè rediksyon an grav tisi yo gen risk pou yo pa jwenn ase oksijèn ak eleman nitritif. Kounye a nou konnen ke plak sa yo ki devlope yo ka kraze oswa kase kò a answit eseye repare sit la epi fòme yon boul san ki ka bloke sikilasyon san nèt..Si sa rive epi sikilasyon san pa retabli tisi yo ka mouri sa yo konsidere kòm yon atak kè.

Kò nou souvan avèti nou ke kè a pa jwenn ase oksijèn ak eleman nitritif. Siy maladi kè kardyovaskulèr yo pral depann de gravite maladi a. Gen kèk moun ki gen CHD pa gen okenn sentòm. Gen kèk ki gen etap nan twò grav doulè nan pwatrin oswa anjin ak kèk gen plis doulè nan pwatrin. Lè sentòm yo prezan, chak moun ka santi yo yon fason diferan. Sentòm maladi atè kowonè ka genyen lou, sere, presyon ak doulè nan pwatrin lan dèyè doulè nan zo tete gaye nan bra yo, zepòl, machwè, kou, ak oswa tounen. Souf kout, feblès ak fatig, kè plen oswa vomisman ka rive osi byen ke ou konnen faktè risk ou yo trè enpòtan epi kontwole risk ou genyen pou fè yon atak kè nan lavni.. Faktè risk yo enkli segondè LDL kolestewòl ke yo rele tou move kolestewòl nivo trigliserid wo ak ba HDL kolestewòl ki se bon kolestewòl tansyon wo ou konnen tou kòm tansyon wo.

Ki prekosyon yo ye?

Li enpòtan pou kontwole tansyon ou sou yon baz regilye lè l sèvi avèk yon manchèt otomatik ka trè senp epi itil. Li enpòtan tou pou kontwole batman kè w lè w inaktif fizikman ogmante risk ou genyen pou w devlope plis maladi kè apre w fin fimen epi w ap twò gwo.. Li enpòtan pou swiv yon rejim ki ba nan grès satire ak ki gen anpil fib fwi ak legim. Asire w ke sik nan san ou kontwole regilyèman si w gen dyabèt epi trete sik nan san ki wo nan objektif rekòmande kontwole faktè risk se kle nan anpeche maladi ak lanmò soti nan. COPD.Asire w ke ou pale ak doktè ou si ou gen nenpòt nan faktè risk sa yo. Gen plizyè fason pou adrese ak trete faktè risk sa yo. Chak moun ta dwe gen yon plan endividyèl pou pran swen risk li yo.

Si w gen nenpòt kesyon konsènan nenpòt nan domèn sa yo, kontakte biwo doktè w la oswa retounen nan enfòmasyon yo bay nan egzeyat ou a. .

Also, heart attacks which are medically referred to as myocardial infarctions occur as a result of many factors often involving some combination of lifestyle, genetics and underlying health conditions. Some important causes of myocardial infarction include:

Coronary Artery Disease (CAD)

The most common cause of heart attacks is coronary artery disease occurs when the arteries that supply blood to the heart get narrowed or blocked due to the build-up of plaques (ateroskleroz). This blocks oxygenated blood from flowing normally in the muscle leading to lack of oxygen and this may lead to damage or death of the heart.

High Blood Pressure (Hypertension)

Over time, chronic high blood pressure can increase the chances of atherosclerosis heart failure and subsequent occurrence of heart attack because it strains both the heart and blood vessels.

High Cholesterol

Higher LDL cholesterol levels and triglycerides might result into arteriosclerosis in where arteries become narrow thereby increasing probability for myocardial infarction.

Fimen

Chemicals in tobacco smoke that are harmful to the blood vessels cause inflammation and speed up atherosclerosis. On the other hand, smokers have much higher rates of suffering from heart attack compared to non-smokers.

Diabetes

Uncontrolled diabetes damages blood vessels and nerves, putting sufferers at risk of atherosclerosis, tansyon wo, and heart attacks. In comparison with patients without diabetes, those diagnosed with diabetes have increased chances of developing heart disease.

Obesity and Sedentary Lifestyle

Increased body weight or obesity and leading sedentary lifestyles are linked to an augmented likelihood of heart disease as well as heart attack cases. Hypertension, diabetes mellitus and hyperlipidemia are caused by both physical inactivity & excess body weight that can also lead to myocardial infarction.

Family History and Genetics

If there has been any family history associated with any heart diseases or heart attacks then it raises their chances indicating that they inherited them genetically. Most importantly, someone can inherit a condition such as familial hypercholesterolemia which will also make him/her more likely to have a stroke.

Stress and Mental Health

Cardiovascular health is adversely affected by long-term pressure, nervousness and depression due to high blood pressure, inflammation and negative coping mechanisms like smoking or eating too much that promote the occurrence of heart disease.

Unhealthy Diet

High intake of fats, salts and refined sugars leads to increased risk obesity, high cholesterol levels and hypertension which are all factors causing heart attacks.

Excessive Alcohol Consumption

Blood pressure raising, increase in triglycerides level and obesity increase the chances for a person getting coronary artery disease (CAD) and directly predisposing them to a heart attack.

It is worth noting that many risk factors for myocardial infarction is preventable by lifestyle changes and medical intervention. A healthy lifestyle that involves regular physical activities, balanced nutrition, quitting smoking, stress control as well as effective management of chronic conditions such as diabetes mellitus and hypertension can greatly reduce the chance of developing myocardial infarction and enhance overall cardiovascular health. Early detection as well as management of risk factors can also be achieved through regular checkups with physicians.